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In females, this same action occurs, except LH and FSH will trigger the release of eggs from from the ovaries (known as follicular rupture), which would lead to increased chances of contraception. This total process in females is known as the HPOA (Hypothalamic Pituitary Ovarian Axis). Much like Nolvadex, Promifen (Clomiphene Citrate) is what would be considered an Estrogen that acts as a ‘fake’ Estrogen in areas of the body, such as breast tissue (so as to block the effects of ‘real’ Estrogen, while it also works as an actual Estrogen in other areas of the body. This is another manner of describing the agonistic/antagonistic nature of Promifen (Clomiphene Citrate) in regards to Estrogen. For anabolic steroid using athletes and bodybuilders that are for the most part males, Promifen (Clomiphene Citrate) can serve as an effective anti-Estrogen for the purpose of mitigating one particular unwanted side effect of Estrogen as a result of the use of aromatizable anabolic steroids (such as Testosterone, Dianabol, Boldenone, etc. This particular unwanted side effect is that of gynecomastia, which refers to the development of breast tissue resulting from excess Estrogen levels in body. Because of Promifen (Clomiphene Citrate)’s Estrogen antagonistic effects on the hypothalamus resulting in the increased production of gonadotropins LH and FSH, Promifen (Clomiphene Citrate) can effectively be utilized to increase endogenous Testosterone production in males. This is especially important for anabolic steroid using individuals that wish to restore proper hormonal function during the weeks following the conclusion of an anabolic steroid cycle, a period known as PCT (Post Cycle Therpay). The traditional protocol for such a use is that of a combination of Nolvadex, Promifen (Clomiphene Citrate), and HCG (Human Chorionic Gonadotropin) for the duration of several weeks, and although much better more modern protocols have been developed, this long-standing traditional PCT protocol is still effective. The use of Promifen (Clomiphene Citrate) for this purpose will be covered in further detail under the Promifen (Clomiphene Citrate) doses section of this profile. All of the tripohenylethylene compounds under the SERM family (Nolvadex, Promifen (Clomiphene Citrate), and Toremifene) also exhibit Estrogen agonistic effects in the liver, meaning the liver is one such area of the body where SERMs such as Promifen (Clomiphene Citrate) (Clomiphene Citrate) will act as an Estrogen rather than block Estrogenic activity there. In studies, this has shown to be a beneficial aspect of SERMs, as Estrogens as well as Estrogen agonists (such as Promifen (Clomiphene Citrate) and Nolvadex) impart a positive effect on cholesterol values through actions in the liver. This is the reason as to why the reduction of Estrogen levels through the use of an aromatase inhibitor (AI) is not always the best decision, as the reduction in Estrogen often results in negatively impacted cholesterol profiles as demonstrated anecdotally as well as in clinical studies. Promifen (Clomiphene Citrate) (Clomiphene Citrate) is a non-steroidal selective Estrogen receptor modulator (SERM) that possesses both mixed agonistic as well as antagonistic properties in relation to Estrogen in different areas of the body. Promifen (Clomiphene Citrate) belongs to a family of compounds known as triphenylethylene compounds, of which Nolvadex (Tamoxifen Citrate) is also a member of, and a very closely related compound to Promifen (Clomiphene Citrate). It has already been established that Promifen (Clomiphene Citrate), being a SERM, does not reduce circulating Estrogen levels in the body, but instead serves to occupy the receptor sites in breast tissue so that Estrogen itself cannot bind to these receptors due to Promifen (Clomiphene Citrate)’s stronger binding strength to it. In layman’s terms, Promifen (Clomiphene Citrate) essentially acts as a ‘fake’ Estrogen that acts as a placeholder at the receptor sites in breast tissue. As a result, Estrogen cannot activate gene transcription in the cells there in order to formulate gynecomastia, and any existing Estrogen that has already bound to receptor sites will essentially be ‘forced’ out of the receptor sites by Promifen (Clomiphene Citrate) which then occupies the receptor site instead. However, it must be noted that Promifen (Clomiphene Citrate)’s action in this area is far weaker and less efficient than its close relative compound Nolvadex, which serves as a better choice for this purpose. Although Promifen (Clomiphene Citrate) could indeed be utilized by anabolic steroid using individuals that wish to prevent or eliminate and reverse formulating gynecomastia in its early stages, its more effective and promising role is that of an endogenous Testosterone production stimulating compound. This is the primary purpose and function of Promifen (Clomiphene Citrate) among anabolic steroid using athletes and bodybuilders, and is the primary desired effect in this sense. It must be noted, however, that research has also demonstrated various advantages that Nolvadex does possess over Promifen (Clomiphene Citrate) in this sense as well, which will be further covered in the next section of this profile. As a SERM, Promifen (Clomiphene Citrate) will not serve to block or reduce any other Estrogenic side effects, however, as it serves only to block Estrogenic activity at the breast tissue area (when major Estrogenic side effects are concerned). Promifen (Clomiphene Citrate) does not (nor do any SERMs) serve to reduce bloating, water retention, rising blood pressure (as a result of water retention), or acne formation ‘ these are all side effects resultant from increasing blood plasma Estrogen levels. 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